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TB-500 Peptide – COA Verified Thymosin Beta-4 Analog for Tissue Repair Research | Not Labs
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TB-500 Peptide (5mg / 10mg)

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TB-500 peptide (5mg, 10mg) — COA-verified thymosin beta-4 analog investigated for its role in tissue repair, angiogenesis, and recovery pathways. High-purity, research-only quality from Not Labs.

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Overview

TB-500 peptide is a synthetic fragment of thymosin beta-4, a naturally occurring protein studied for its role in cell migration, tissue regeneration, and angiogenesis. Researchers examine TB-500 for its potential to accelerate wound closure, promote vascular growth, and support tissue remodeling in controlled laboratory models. As a focused research compound, TB-500 allows investigation of thymosin beta-4’s core bioactive region without the complexity of the full-length protein.

Compound Breakdown

TB-500: Derived from the amino acid region of thymosin beta-4 most associated with biological activity. Preclinical data suggest TB-500 interacts with actin—the protein central to cytoskeletal structure and cellular motility. By influencing actin polymerization, TB-500 may enhance cellular migration into damaged areas and contribute to repair processes.

Key Findings in Research Models

  • Actin Regulation → TB-500 has been observed to regulate actin binding, supporting wound closure and structural recovery in preclinical systems.
  • Angiogenesis → Studies indicate TB-500 may promote new blood vessel formation, improving nutrient delivery and tissue remodeling.
  • Tissue Regeneration → Research suggests TB-500 influences extracellular matrix stabilization and collagen deposition in laboratory models.
  • Systemic Distribution → Animal studies report broad tissue distribution, allowing researchers to assess multi-organ effects.

Research Applications

  • Wound Healing → studied for accelerated closure rates and tissue integrity in animal models.
  • Cardiac Research → investigated for potential repair activity following ischemic injury.
  • Musculoskeletal Models → evaluated for tendon, ligament, and muscle regeneration.
  • Angiogenesis Studies → explored for stimulation of vascularization and recovery pathways.

While laboratory results are promising, findings remain limited to experimental models and do not imply any clinical outcomes.

Related Categories

TB-500 is included in the Muscle & Physique and Tissue & Recovery categories. It is often studied in conjunction with BPC-157 and GHK-Cu in connective tissue and regeneration research.

Compliance Statement

All Not Labs products are strictly intended for research use only. TB-500 peptide is not approved for human or veterinary use.

Certificate of Analysis

FAQ

  • How stable is TB-500 peptide under laboratory conditions?
    Lyophilized TB-500 peptide is typically stable at -20°C. After reconstitution, it may remain stable for several weeks under refrigeration if aliquoted to prevent repeated freeze-thaw cycles.

  • What laboratory applications commonly investigate TB-500?
    TB-500 has been studied in wound healing models, angiogenesis research, musculoskeletal repair, and cardiac recovery following ischemic injury.

  • How does TB-500 compare to full-length thymosin beta-4?
    TB-500 represents the active region of thymosin beta-4, enabling researchers to study the most relevant sequences without the complexity of the full protein.

  • What do studies suggest about TB-500’s systemic distribution?
    Animal research indicates TB-500 disperses widely across tissues, allowing investigators to assess multi-organ responses in recovery models.

  • What compounds are often studied alongside TB-500?
    Researchers frequently examine TB-500 in combination with BPC-157 and other regenerative peptides to explore potential synergy in tissue repair studies.

  • Why is COA verification important for TB-500 research?
    COA verification ensures that every vial of TB-500 peptide from Not Labs meets rigorous standards for purity and reproducibility, giving researchers confidence in their data.

References

References:

  1. Sun Q, et al. (2023). Thymosin beta-4 alleviates cardiac damage in pressure overload-induced heart failure via regulation of the NRG-1/ErbB pathway. Frontiers in Cardiovascular Medicine. PubMed
  2. Wang J, et al. (2016). Thymosin beta-4 promotes angiogenesis and cardiac regeneration after myocardial infarction in mice. Journal of Cellular and Molecular Medicine. PubMed
  3. Shao Y, et al. (2020). Thymosin beta-4 promotes corneal epithelial wound healing and reduces inflammation in a mouse alkali burn model. Experimental Eye Research. PubMed
  4. Hu J, et al. (2023). Thymosin beta-4 attenuates pulmonary fibrosis by regulating epithelial–mesenchymal transition and collagen deposition in vivo. International Journal of Molecular Sciences. PubMed

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